Dissection of the immunomodulatory effects of ES-62 on inflammatory autoimmune disease

M.A. McGrath; D.E. Kean; I.B. McInnes; W. Harnett; M.M. Harnett

doi:10.1111/j.1365-2567.2008.02974.x

Dissection of the immunomodulatory effects of ES-62 on inflammatory autoimmune disease

M.A. McGrath, D.E. Kean, I.B. McInnes, W. Harnett, M.M. Harnett

Strathclyde Institute Of Pharmacy And Biomedical Sciences

Research output: Contribution to journal › Article › peer-review

33 Citations (Scopus)

Abstract

The phosphorylcholine-containing glycoprotein ES-62, secreted from filarial nematodes, may have therapeutic potential in autoimmune inflammatory disease as it has been shown to possess the ability to selectively inhibit aberrant pro-inflammatory cytokine production without leaving the host immunocompromised.

Original language	English
Pages (from-to)	122-123
Number of pages	1
Journal	Immunology
Volume	125
Issue number	s1
DOIs	https://doi.org/10.1111/j.1365-2567.2008.02974.x
Publication status	Published - Dec 2008

Keywords

immunomodulatory
inflammatory autoimmune disease
ES-62

Access to Document

10.1111/j.1365-2567.2008.02974.x

Cite this

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title = "Dissection of the immunomodulatory effects of ES-62 on inflammatory autoimmune disease",

abstract = "The phosphorylcholine-containing glycoprotein ES-62, secreted from filarial nematodes, may have therapeutic potential in autoimmune inflammatory disease as it has been shown to possess the ability to selectively inhibit aberrant pro-inflammatory cytokine production without leaving the host immunocompromised.",

keywords = "immunomodulatory, inflammatory autoimmune disease, ES-62",

author = "M.A. McGrath and D.E. Kean and I.B. McInnes and W. Harnett and M.M. Harnett",

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AB - The phosphorylcholine-containing glycoprotein ES-62, secreted from filarial nematodes, may have therapeutic potential in autoimmune inflammatory disease as it has been shown to possess the ability to selectively inhibit aberrant pro-inflammatory cytokine production without leaving the host immunocompromised.

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